Yvonne Chaka Chaka found herself on Twitter trending list after she questioned COVID-19 vaccine development.
The 55-year-old legend, has expressed her doubt over vaccine development, she questioned on why it developed so quickly compared to HIV and Malaria.
She Tweeted: “Vaccine, Vaccine. We still don’t have one for Malaria non for HIV, but in a few months we have one for Covid can someone tell me how possible is this @WHO @EU_Partnerships @UN @NEPAD_Agency @PresidencyZA @Davos @unwomenafrica.”
Her comment shocked some followers, as they expressed disappointment of her question.
One user responded: “She’s a UN Goodwill Ambassador married to a doctor. As a longtime admirer, I am so disappointed.”
Another user told Yvonne Chaka Chaka to take a time browsing on internet on her doubt before questioned on social media.
“Sigh… Mama Africa, Princess of Africa or whatever figment of imagination title you go by. Please I beg you to take a few minutes off of your studio time and research these viruses respectively on the internet and maybe, just maybe…”
One scholar points the challenges which based on actual facts on why it is hard to develop quickly HIV/Aids or Malaria.
“Malaria is a parasite. HIV attacks the immune system itself. Vaccine technology doesn’t work for these diseases. HIV is managed by ARVs which inhibit viral reproduction. It took millions and years to develop ARVs so your “victim” hypothesis is untrue,” user tweeted.
The development of COVID-19 vaccine swirling different conspiracies, many are still not aware of nature and real facts about these diseases.
According to Verywell Health, The history of HIV vaccine development has been marked by numerous setbacks and disappointments, with each apparent “breakthrough” presenting even more challenges and hurdles to overcome.
Oftentimes it seems that for one step forward researchers take, an unforeseen obstacle sets them back by one and even two steps.
The efforts to develop an HIV vaccine have been hampered by the genetic diversity of the virus itself. The replication cycle of HIV is not only fast (a little more than 24 hours) but is prone to frequent errors, churning out mutated copies of itself which recombine into new strains as the virus is passed from person to person.
Secondly, fighting HIV demands a robust response from the immune system, and this again where systems fail. Traditionally, specialized white blood cells called CD4 T-cells initiate the response by signaling killer cells to the site of the infection. Ironically, these are the very cells that HIV targets for infection. By doing so, HIV hobbles the body’s ability to defend itself as the CD4 population is systematically depleted, resulting in the eventual breakdown of defenses called immune exhaustion.
Finally, the eradication of HIV is thwarted by the virus’ ability to hide from the body’s immune defenses. Soon after infection, while other HIV is circulating freely in the bloodstream, a subset of virus (called provirus) embeds itself in hidden cellular sanctuaries (called latent reservoirs). Once inside these cells, HIV is shielded from detection.
Based on Verywell Health facts, there is more research and experiment needed to understand the HIV virus and infection which could take many years for scientists to accomplish the vaccine.